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#Celltherapy Reel by @mdandersoncancercenter (verified account) - Cancer cells don't act alone.

They communicate with each other and with the immune system, shaping how tumors grow and how patients respond to treat — Cancer cells don’t act alone. They communicate with each other and with the immune system, shaping how tumors grow and how patients respond to treatment. At MD Anderson, researchers are decoding this cellular language to better understand tumor behavior and improve immunotherapy for cancer patients.

#Celltherapy Reel by @dr.hasanmorcali (verified account) - Latest Breakthroughs in Cancer Research and Treatment (2026)

🧬 Towards a Universal mRNA Cancer Vaccine
Researchers have demonstrated that a generali — Latest Breakthroughs in Cancer Research and Treatment (2026) 🧬 Towards a Universal mRNA Cancer Vaccine Researchers have demonstrated that a generalized mRNA approach can stimulate a broad anti-tumor immune response rather than targeting a single cancer type. 🧪 Next-Generation Immunotherapies: Personalized and Tumor-Specific Targeting Advanced immunotherapies now identify tumor-specific antigens to generate more precise and durable immune responses, improving outcomes in cancers like melanoma and lung cancer. 🧫 Expanding CAR-T Cell Therapy into Solid Tumors CAR-T therapies, once limited to blood cancers, are now showing promising results in solid tumors such as pancreatic, lung, and brain cancers. 🧬 Liquid Biopsy Revolution in Cancer Detection Highly sensitive blood-based tests can detect circulating tumor DNA, enabling early diagnosis and guiding personalized treatment decisions across multiple cancer types. ⚡ FLASH Radiotherapy: Ultra-Fast and Safer Treatment FLASH radiotherapy delivers high-dose radiation in milliseconds, effectively controlling tumors while significantly reducing damage to surrounding healthy tissues. 💊 Antibody-Drug Conjugates: Smart Targeted Cancer Therapy ADC therapies combine monoclonal antibodies with cytotoxic drugs to selectively deliver treatment to cancer cells, increasing efficacy while minimizing side effects. For more detailed information, please contact us Asst. Prof Dr. Hasan MORCALI

#Celltherapy Reel by @annetbondar1 - A cancer epidemic. What's the cause? — A cancer epidemic. What’s the cause?

#Celltherapy Reel by @immunotherapyinstitute - Most cancer conversations focus on the tumor.

Very few focus on the terrain.

Cancer doesn't grow in isolation.
It interacts with the immune system. — Most cancer conversations focus on the tumor. Very few focus on the terrain. Cancer doesn’t grow in isolation. It interacts with the immune system. It adapts to pressure. It reshapes its environment. At the Immunotherapy Institute, we specialize in combining advanced targeted therapies with autologous immunotherapy — including dendritic cell and NK cell strategies — layered alongside conventional treatment. Not to replace standard-of-care. But to expand it. Because aggressive disease requires multi-angle strategy. The question isn’t whether conventional oncology works. The question is whether your strategy is complete.#Immunotherapy #CancerImmunotherapy #IntegrativeOncology #AdvancedCancerCare #Stage4Warrior #CancerResearch #TargetedTherapy #NKCells #DendriticCells #CancerAwareness #OncologyCare #CancerEducation #CancerAdvocate #PrecisionMedicine #ImmuneBasedTherapy

#Celltherapy Reel by @iylononcology - 𝐇𝐨𝐰 𝐂𝐚𝐧𝐜𝐞𝐫 𝐂𝐞𝐥𝐥𝐬 𝐇𝐢𝐝𝐞 𝐟𝐫𝐨𝐦 𝐭𝐡𝐞 𝐈𝐦𝐦𝐮𝐧𝐞 𝐒𝐲𝐬𝐭𝐞𝐦?

Cancer does not develop overnight. As cells change and grow, some — 𝐇𝐨𝐰 𝐂𝐚𝐧𝐜𝐞𝐫 𝐂𝐞𝐥𝐥𝐬 𝐇𝐢𝐝𝐞 𝐟𝐫𝐨𝐦 𝐭𝐡𝐞 𝐈𝐦𝐦𝐮𝐧𝐞 𝐒𝐲𝐬𝐭𝐞𝐦? Cancer does not develop overnight. As cells change and grow, some acquire abilities that help them avoid the body’s natural immune defenses. One important mechanism behind this is known as genetic immune escape, a process in which cancer cells develop genetic changes that make them less visible to the immune system. These changes can occur early as normal cells begin transforming into cancer. Over time, cells that are better at avoiding immune detection are more likely to survive and multiply, allowing tumors to grow and spread. Researchers are studying how these genetic changes interact with the tumor’s surrounding environment to help cancers evade immune responses. Understanding this process is important for precision oncology, as it can guide the development of better methods for early cancer detection and more targeted treatments. To learn more about how genetic immune escape influences precision oncology, watch the video linked below. https://youtube.com/shorts/3XNJU_zJSA8?feature=share 𝘈𝘵 𝘐𝘺𝘭𝘰𝘯 𝘗𝘳𝘦𝘤𝘪𝘴𝘪𝘰𝘯 𝘖𝘯𝘤𝘰𝘭𝘰𝘨𝘺 (www.iylon.com), 𝘸𝘦 𝘩𝘢𝘳𝘯𝘦𝘴𝘴 𝘨𝘦𝘯𝘦𝘵𝘪𝘤 𝘪𝘯𝘴𝘪𝘨𝘩𝘵𝘴 𝘵𝘰 𝘱𝘳𝘰𝘷𝘪𝘥𝘦 𝘱𝘦𝘳𝘴𝘰𝘯𝘢𝘭𝘪𝘻𝘦𝘥, 𝘦𝘷𝘪𝘥𝘦𝘯𝘤𝘦-𝘣𝘢𝘴𝘦𝘥 𝘳𝘦𝘤𝘰𝘮𝘮𝘦𝘯𝘥𝘢𝘵𝘪𝘰𝘯𝘴, 𝘩𝘦𝘭𝘱𝘪𝘯𝘨 𝘨𝘶𝘪𝘥𝘦 𝘵𝘩𝘦 𝘳𝘪𝘨𝘩𝘵 𝘮𝘦𝘥𝘪𝘤𝘪𝘯𝘦, 𝘧𝘰𝘳 𝘵𝘩𝘦 𝘳𝘪𝘨𝘩𝘵 𝘱𝘢𝘵𝘪𝘦𝘯𝘵, 𝘢𝘵 𝘵𝘩𝘦 𝘳𝘪𝘨𝘩𝘵 𝘥𝘰𝘴𝘦, 𝘢𝘵 𝘵𝘩𝘦 𝘳𝘪𝘨𝘩𝘵 𝘵𝘪𝘮𝘦, 𝘢𝘯𝘥 𝘢𝘪𝘮𝘦𝘥 𝘢𝘵 𝘵𝘩𝘦 𝘳𝘪𝘨𝘩𝘵 𝘵𝘢𝘳𝘨𝘦𝘵. @senduraimani @jtarantino73

#Celltherapy Reel by @scientific_american (verified account) - Personalized cancer vaccines based on mRNA are showing real promise by training a patient's immune system to recognize and attack the unique mutations — Personalized cancer vaccines based on mRNA are showing real promise by training a patient’s immune system to recognize and attack the unique mutations in their own tumor. Early trials have kept some patients with aggressive cancers, including pancreatic cancer, in long-term remission, hinting at a major shift in how cancer could be treated after surgery. But just as dozens of these vaccines move through clinical trials worldwide, U.S. funding cuts and political hostility toward mRNA technology are putting this research at risk. Read the December cover story at the link in our bio. ✏️🎞️🎤: Tom Lum (@tomlumperson) 📝: Rowan Moore Gerety

#Celltherapy Reel by @cancervaxbiotech - What if cancer treatment could attack only cancer cells - and leave healthy cells untouched? That's exactly what this breakthrough is about.
For years — What if cancer treatment could attack only cancer cells — and leave healthy cells untouched? That’s exactly what this breakthrough is about. For years, one of the biggest challenges in medicine hasn’t just been creating powerful therapies — it’s been delivering them precisely where they need to go. Here’s what makes this different: 🧬 A targeted nanoparticle delivery system designed to hit the right cells 🎯 A Smart mRNA payload that knows where it’s supposed to go 🚫 Healthy cells left unharmed 🔬 Proven results in laboratory (in vitro) testing 🐭 Now advancing to animal studies and beyond In simple terms? We’ve reduced the risk. We know the therapy reaches the cancer cell — and not the healthy ones. If this continues to succeed, it could represent a major shift in how cancer treatments are developed. #CancerResearch #CancerBreakthrough #mRNA #Nanotechnology #Biotech #Oncology

#Celltherapy Reel by @imelda_j - scans have a limit. small cancer cells can't always be seen. after surgery, the risk isn't what shows up on scans, but what may be left behind. micros — scans have a limit. small cancer cells can’t always be seen. after surgery, the risk isn’t what shows up on scans, but what may be left behind. microscopic cancer cells can still remain. personalised vaccines help the immune system clear what scans can’t detect. that’s why this approach can reduce relapse. 🙂 cancer touches many lives, but it’s often explained in complicated ways. after 15 years in cancer innovation, i care about sharing it simply and clearly. follow for cancer updates in simple english. @imelda_j take care of your heart,🤍 papers: adjuvant mRNA-4157/v940 plus pembrolizumab versus pembrolizumab alone in resected high-risk melanoma. keynote-942 (phase 2b, randomised). the lancet, 2023. ~44–49% relative reduction in recurrence. ott et al. an immunogenic personal neoantigen vaccine for patients with melanoma and other solid tumors. nature, 2017. extended follow-up showed 0/9 recurrences at ~40 months. rojas et al. personalized rna neoantigen vaccines stimulate t cells in pancreatic cancer. nature, 2023. vaccine-specific t-cell responses in ~50% of patients, with responders showing delayed recurrence.

#Celltherapy Reel by @biochemedits - what's your favorite type of T cell? source footage: Peter MacCallum Cancer Centre CAR-T Cells: Engineered Cancer Killers, City of Hope CAR T Cell The — what's your favorite type of T cell? source footage: Peter MacCallum Cancer Centre CAR-T Cells: Engineered Cancer Killers, City of Hope CAR T Cell Therapy Explained (Animation) | City of Hope. review by Sun, L., Su, Y., Jiao, A. et al. T cells in health and disease. Signal transduction and targeted therapy 8, 235 (2023). "T lymphocytes (T cells) are the major cell components of the adaptive immune system, responsible for mediating cell-based immune responses to keep the host healthy and prevent various types of diseases. T cells are developed from bone marrow (BM)-derived thymocyte progenitors in the thymus, and broadly grouped into CD4+ and CD8+ αβ T cells in addition to rear populations of γδ T cells and NKT cells. αβ T cells recognize antigens that are presented by major histocompatibility complex (MHC) molecules on antigen-presenting cells (APCs). Upon recognition of cognate antigens (signals 1) by T cell receptor (TCR) and costimulatory molecules (signals 2) on APCs, and cytokines (signals 3), naïve CD4+ and CD8+ T cells undergo activation, clonal expansion, and differentiation to execute their effector functions of destroying infected cells, producing cytokines and regulating immune responses. A small population of T cells develops into memory T cells which exhibit rapid effector functions upon reencountering the same antigens and provide the host with potent and long-term protection. In parallel, there exists a subpopulation of CD4+ T cells, named regulatory T (Treg) cells, that maintain peripheral immune tolerance." #immunology #immunecells #cancer #tcells #cityboy

#Celltherapy Reel by @tomasz.grzywa (verified account) - 🔬 Why do CAR-T cells struggle in solid tumors?

CAR-T therapy has shown remarkable success in blood cancers, but solid tumors remain a major challeng — 🔬 Why do CAR-T cells struggle in solid tumors? CAR-T therapy has shown remarkable success in blood cancers, but solid tumors remain a major challenge. One of the key barriers is the tumor microenvironment, which actively suppresses immune responses. A major player in this suppression is tumor-associated macrophages (TAMs). Macrophages normally play essential roles in our body - clearing pathogens, removing dead cells, and coordinating immune responses. But tumors have learned how to reprogram these cells to work against us. Inside tumors, macrophages often adopt a pro-tumor phenotype that supports tumor growth, promotes tissue remodeling and angiogenesis, and suppresses anti-tumor immune responses, including CAR-T cell activity. One important inhibitory receptor involved in this process is LAIR1. 🎥 What you’re seeing in this video This experiment uses a live 3D tumor model with real-time imaging. All immune cells were derived from the same human PBMC donor and fluorescently labeled: 🔴 CAR-T cells 🟢 tumor-associated macrophages 🔵 CD70⁺ glioblastoma tumor cells ⚠️ What happens in the control condition When macrophages are present, CAR-T cells become suppressed. Their migration is impaired, they interact less with tumor cells, and tumor killing is strongly reduced (first part of the reel!) This demonstrates how TAMs can act as a physical and functional barrier to T-cell activity. 💡 What happens when LAIR1 is blocked Adding an antibody targeting LAIR1 (aLAIR1) disrupts macrophage-mediated inhibition. As a result CAR-T cells move more freely, they engage tumor cells more frequently, and tumor killing increases 🧠 Take-home message Tumors hijack macrophages to suppress immune attack and protect themselves. Targeting myeloid checkpoints like LAIR1 may help unlock the full potential of CAR-T therapy in solid tumors by reprogramming the tumor microenvironment. Credits: Tao H et al. JCI 2025 #scientist #science #laboratory #immunology #medicine

#Celltherapy Reel by @north.star.cancer.advocacy - Finding immunotherapy targets that are ONLY expressed on cancer cells and NOT on healthy tissue is extremely challenging.

So a company called A2 Bio — Finding immunotherapy targets that are ONLY expressed on cancer cells and NOT on healthy tissue is extremely challenging. So a company called A2 Biotherapeutics is designing what’s called Boolean logic-gated CAR-T cellular immunotherapies. A lot of cancers overexpress EGFR but because it’s also expressed on healthy tissue it can be a challenging target. A2 Bio’s Denali CAR-T trial is available to people with metastatic Triple Negative Breast Cancer. These CAR-T are designed to only attack EGFR-expressing cells if the cell is ALSO NOT expressing its HLA-A*02 molecule (HLA and MHC are the same thing but in humans we call it HLA). A2 Biotherapeutics has treated many people with this Boolean CAR-T strategy and at least one person with lung cancer had a complete response with their Mesothelin (MSLN) targeting CAR-T. CAR-T have rarely provided complete responses in solid tumor trials so this was a huge milestone. However an IG friend of mine was the first person treated with A2 Bio’s EGFR-targeting CAR-T and she passed away approximately 6 weeks later. The trial, to the best of my knowledge, was not put on an FDA-hold following her death but it’s important to know that these CAR-T trials are both cutting-edge and also carry significant risk. I always recommend people inquire about clinical trial related deaths. To qualify for the Denali trial, you first have to be screened through their Basecamp trial which assesses whether: 1) your healthy tissue normally expresses the HLA-A*02 type and 2) whether your cancer cells express EGFR and have lost the gene which codes for HLA-A*02 In order to qualify for the Denali trial, all three criteria have to be met in the Basecamp screening. If this type of proof of concept trial proves successful, the product could be extended to people with other HLA-A types. Some tumor genomics companies are now providing people with their basic HLA typing but the Basecamp trial would still be worth applying to to find out your cancer’s EGFR expression levels and whether your tumor(s) have lost one of their HLA-A genes. Basecamp screening could be done well in advance of needing other options. #stageivneedsmore #tnbc #tnbcthriver

#Celltherapy Reel by @immunotherapyinstitute - Cancer care isn't black and white.

It's not conventional vs alternative.
It's complete vs incomplete.

At the Immunotherapy Institute, we specialize — Cancer care isn’t black and white. It’s not conventional vs alternative. It’s complete vs incomplete. At the Immunotherapy Institute, we specialize in combining advanced targeted therapies with autologous immunotherapy — including dendritic cell and NK cell strategies — layered alongside conventional treatment. We believe patients deserve access to expanded options, strategic immune integration, and personalized 1:1 physician-led care — especially in advanced and recurrent cases. Not rebellion. Not replacement. Expansion. Because cancer is adaptive. And strategy should be too. — If this challenges how you’ve been thinking about treatment, that’s the point.

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